Natural Remedy for Elevated Blood Sugar

horseradish root

Blood Sugar
When the findings are disappointing, and your doctor will ask whether you want to start insulin therapy or try a diet to reduce blood sugar. Many succeeded to heal and to maintain normal blood sugar levels, using this natural remedy.

Ingredients

  •  A horseradish root, medium size (about 20 cm long, 2 cm wide)
  •  One clove garlic
  •  2 beers

Preparation
Wash the horseradish root nice several times. Leave to stand for 15-20 minutes in water in which you have added a cup of malic acid. Don’t peel it, chop finely or grind it. Along with finely chopped garlic place them into a larger jar and pour the beer. Close the jar and store it for 10 days in a dark place. Then strain and use as follows:

  • The first two days, one teaspoon three times a day.
  • Further use one tablespoon three times a day.
  • Treatment is for one month.

In addition, you should drink 150 ml oatmeal water, 4 times a day, properly arranged.
(50 g oatmeal boil over on low heat for 15 minutes, then let it stand for 1 hour. Grains are squeezed, then strained through gauze).

Source: healthy tavern


Diabetes in Middle Age May Cause Memory Problems

People who develop type 2 diabetes or high blood pressure in middle age appear more likely to suffer brain damage that can contribute to dementia as they grow older, a new study finds.

Diabetes might actually shrink the brain over a long period of time, reducing the size of crucial areas like the hippocampus, which plays an important role in short- and long-term memory, according to the study.

Additionally, diabetes and high blood pressure both seem to increase a person’s risk of micro-strokes and other damage to the blood vessels that feed the brain, the study authors said.

“People who had diabetes earlier in life had much worse brain [structure] than those who had it later in life,” said lead author Dr. Rosebud Roberts, a Mayo Clinic researcher. “These scans are showing us that cognitive impairment happens over a long period of time. The earlier you develop type 2 diabetes, the more likely you are to have damage.”

Diabetes has long been linked to problems with thinking and memory later in life, but this study is the first to provide solid evidence explaining why that occurs, said Keith Fargo, director of scientific programs and outreach for the Alzheimer’s Association.

“We are very excited about this study,” Fargo said. “It has been known for quite some time that there is some kind of link between diabetes and cognitive ability later in life. What has not been known yet is why this link exists and how it develops over time.”

The study involved more than 1,400 people with an average age of 80, according to the report published online March 19 in the journal Neurology. The study participants had at most slight memory and thinking problems called mild cognitive impairment. The researchers assessed the participants’ thinking and memory skills, noting any signs of mild impairment.

The study participants then underwent MRI brain scans to look for signs of brain damage that can be an early indication of dementia.

Finally, the researchers reviewed the participants’ medical records to see whether they had been diagnosed with diabetes or high blood pressure in middle age, which, for the purposes of this study, ran from 40 to 64.

The study authors found that people who developed diabetes in middle age had brains that were on average 2.9 percent smaller than people who didn’t have diabetes. And their hippocampi were even smaller — an average of 4 percent smaller than those of non-diabetics.

“When your hippocampus begins to shrink, you begin to lose your long-term memory and your ability to remember recent events,” said Roberts, who also is a member of the American Academy of Neurology.

Source: web md


New drug target for controlling high blood sugar discovered

Researchers have identified a new potential therapeutic target for controlling high blood sugar.

Researchers showed that lipid molecules called phosphatidic acids enhance glucose production in the liver. These findings suggest that inhibiting or reducing production of phosphatidic acids may do the opposite.

Senior author Dr. Anil Agarwal, Professor of Internal Medicine, said that their study establishes a role for phosphatidic acids in enhancing glucose production by the liver and identifies enzymes involved in the synthesis of phosphatidic acids as potential drug targets.

These observations were made while studying a mouse model of lipodystrophy, a rare metabolic disease in which the body is devoid of fat. Lipodystrophy patients often develop diabetes and accumulate fat in the liver because of an imbalance in the body’s ability to properly regulate lipids and glucose.

The causal gene, AGPAT2, which is involved in the synthesis of phosphatidic acid and triglycerides, was removed in the mice, resulting in rodents with generalized lipodystrophy. The research team then examined what impact this genetic manipulation had on phosphatidic acids and glucose production.

The buildup of these lipid molecules was due to an increase in the levels of two enzymes in the liver, diacylglycerol kinase and phospholipase D. Researchers also discovered a marked increase in glucose production in the livers of the lipodystrophic mice.

The lack of normal insulin signaling in these lipodystrophic mice led to unrestricted production of phosphatidic acid, Dr. Agarwal explained, contributing to development of hyperglycemia, or high blood sugar.

The study has been published in The Journal of Biological Chemistry.

Source: Business standard